The oculomotor nerve and its associated cranial nerve nuclei exist within the midbrain. The midbrain develops from the mesencephalon. Neuroblasts from the basal plates develop into the tegmentum. The tegmentum includes cranial nerves III and IV, Edinger-Westphal nuclei, oculomotor nuclei, trochlear nuclei, red nuclei, and reticular nuclei A palsy of the 3rd cranial nerve can impair eye movements, the response of pupils to light, or both. These palsies can occur when pressure is put on the nerve or the nerve does not get enough blood. People have double vision when they look in a certain direction, the eyelid droops, and the pupil may be widened (dilated) From Wikipedia, the free encyclopedia Oculomotor nerve palsy is an eye condition resulting from damage to the third cranial nerve or a branch thereof. As the name suggests, the oculomotor nerve supplies the majority of the muscles controlling eye movements Oculomotor nerve palsies, or third nerve palsies, result in weakness of the muscles supplied by the oculomotor nerve, namely the superior rectus, inferior rectus, medial rectus, inferior oblique, and levator palpebrae superioris muscles In contrast, the parasympathetic fibers travel superficially along the medial portion of cranial nerve III. The parasympathetic fibers that mediate miosis start in the Edinger-Westphal nucleus, and their axons track along the periphery of cranial nerve III to synapse with the postganglionic cells in the ciliary ganglion (see Figure 4)
Third (oculomotor) nerve, innervates eyelid muscles and external ocular muscles (except lateral rectus and superior oblique) Nerve also carries parasympathetic fibers on external surface allowing for pupillary constriction Palsy causes diplopia except in lateral gaze (lateral rectus innervated by CN VI Ptosis may be partial or complete. Nuclear lesions that spare the central caudate subnucleus to the levator palpebrae superioris may produce third nerve palsies without ptosis
The ptosis of CN-III (oculomotor nerve) palsy is typically accompanied by an external ophthalmoplegia. The patient may not complain of diplopia if the ptosis is severe enough to occlude the visual axis. Figure 3, above and right. Acute, pupil-involving CN-III palsy. Urgent neuroimaging confirmed intracranial aneurysm Patients with chronic and stable ptosis or ophthalmoplegia due to oculomotor nerve palsy may benefit from reconstructive lid or strabismus surgery. Younge BR. Paralysis of cranial nerves III. RUCKER CW. Paralysis of the third, fourth and sixth cranial nerves. Am J Ophthalmol. 1958 Dec. 46 (6):787-94. . Rucker CW. The causes of paralysis of the third, fourth and sixth cranial nerves. Am J Ophthalmol. 1966 May. 61 (5 Pt 2):1293-8. . Berlit P. Isolated and combined pareses of cranial nerves III, IV and VI Cranial nerve III is the oculomotor nerve. It assists in moving the muscles outside and within the eye. It is a nerve that carries both motor and parasympathetic fibres to assist in our ability to see the environment around us. In this article, we take a closer look at the oculomotor nerve and its course to the eye Incomplete CN-III palsies show partial losses of up-, down-, and medial-gaze, along with partial ptosis with some CN-III-innervate muscles more affected than others. In such cases—as the clinical vignette illustrates—recognition that the patient's ocular misalignment is a form of third-nerve palsy can be challenging
A nerve called cranial nerve 3 (or the 3rd nerve) controls the upper eyelid muscle (levator muscle). When this nerve is damaged, then neurogenic ptosis occurs. Neurogenic ptosis can be the result of a stroke, trauma, or a condition called Horner's syndrome. Treatment of neurogenic ptosis depends on how much residual levator strength is present For example, the pupils may be spared and there may be no ptosis. However, an isolated unilateral mydriasis or isolated unilateral or bilateral adduction deficit (medial rectus palsy) is almost never related to a third nerve palsy. Classification of Third Cranial Nerve Palsies Third nerve palsies may be classified as follows
49 year-old man with a history of multiple myeloma in remission, status post autologous stem cell transplant, presented to the Neuro-Ophthalmology clinic at the University of Iowa Hospitals & Clinics with a 1-2 month history of progressively-increasing diplopia and ptosis of the left upper eyelid. The diplopia was oblique and constant, and resolved upon closing either eye The workup should include neuroimaging. If the results of the neuroimaging are normal, and the findings are variable, myasthenia gravis should be considered, and additional testing should be ordered to assist in the diagnosis. Case report: This case report presents a 3-year-old boy who presented with a sudden onset of ptosis and hypertropia Horner's Syndrome is a disruption of the sympathetic nerve supply to the eye. The involved neurons descend from the brain-stem at the level of C8-T2, ascend in the sympathetic chain over the apex of the lung to the superior cervical ganglion at C3-C4, then pass along the ICA to the cavernous sinus and from here, enter the eye Terson syndrome with cranial nerve 3 palsy due to subarachnoid hemorrhage from arteriovenous malformation and aneurysmal rupture: Subject: terson syndrome, vitreous hemorrhage, oculomotor palsy, cranial nerve 3 palsy, ptosis: Creato
HZO-related ophthalmoplegia is uncommon but can affect cranial nerves III, IV, or V I . Most patients are over the age of 50 years. The ophthalmoplegia usually occurs one to two weeks after the rash. The reported cases of HZO ophthalmoplegia have demonstrated abnormal enhancement of the orbit, cranial nerves, optic nerve, and pons . To our. Third (Occlulomotor) Cranial Nerve Palsy Signs and symptoms: Pt usually present with sudden onset unilateral ptosis. Rarely, bilateral if the damage occurs to the third nerve nucleus, frequently accompanied by peri orbital pain or headache. Pt may or may not complains of double vision because the ptosis obscures the vision in the affected eye H49.00 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2021 edition of ICD-10-CM H49.00 became effective on October 1, 2020. This is the American ICD-10-CM version of H49.00 - other international versions of ICD-10 H49.00 may differ
A 64-year-old man presented with a 2-day history of acute onset painless left ptosis. He had no other symptoms; importantly pupils were equal and reactive and eye movements were full. There was no palpable mass or swelling. He was systemically well with no headache, other focal neurological signs, or symptoms of fatigue. CT imaging showed swelling of the levator palpebrae superioris suggestive. Advertisement. It is the sympathetic nerve that is affected along with the 6th cranial nerve. This is due to a break in the sympathetic nerves going to the eyes. Horner's syndrome can occur spontaneously or due to other illnesses. For example, it can be caused by a brain, head, neck, or spinal cord diseases, such as 3rd , 4th, 6th cranial nerves:- The Oculomotor, Trochlear, Abducent Nerves. Action- Control all external ocular muscles and elevators of lid. Examination- Ptosis- • Note for any Ptosis. Note whether it is unilateral or bilateral, constant or variable . The eye may also be in abduction and turned down. If the visceral component is impaired, the papillary reflex is lost and the pupil is dilated. A lesion in the oculomotor nerve may also cause double vision (diplopia) or a blown pupil—a pupil that cannot.
Common cranial nerve examination questions for medical finals, OSCEs and MRCP PACES: oculomotor (3rd), trochlear (4th) + abducens (6th) Click on the the questions below to see the answers, or click here for questions about other cranial nerves and click here to learn how to examine the cranial nerves. Question 1: Question 2: Question 3: Question 4: [ Neurogenic ptosis is caused by cranial nerve III palsies, Horner's syndrome, Marcus-Gunn jaw-winking ptosis, or cranial nerve VII palsies. Palsies of cranial nerve III can result from several conditions including aneurysms, compressive mass within the cavernous sinus, diabetes, and ischemia Anatomy of 3rd cranial nerve. 1. ANATOMY OF THIRD CRANIAL NERVE OPTOM FASLU MUHAMMED. 2. The cranial nerves 12 in number. Are part of the peripheral nervous system. The craniocaudal sequence of cranial nerves is as follows • 1- Olfactory • 2- Optic • 3- Oculomotor • 4- Trochlear • 5- Trigeminal • 6- Abducent • 7- Facial • 8. Congenital Ptosis Congenital Third Nerve Palsy. Third nerve palsies that are present at birth can be due either to a developmental abnormality or intrauterine/birth trauma. They represent nearly half of third-nerve palsies seen in children. Infants usually present with unilateral ptosis, some amount of ophthalmoplegia and pupil involvement.
There are 12 pairs of cranial nerves although the optic nerve is really an extension of the brain rather than a peripheral nerve. The ability to test them swiftly, efficiently and to interpret the findings should be a core competency for general practice Ipsilateral cranial nerve (CN) dysfunction involving any one or combination of CN III, IV, V1, V2, and VI, sympathetic fibers (i.e., Horner syndrome) suggests cavernous sinus lesion especially cranial nerve V and sympathetic fiber involvement localizes specifically to cavernous sinus
Anatomy Cranial nerves III, IV and VI control our extraocular muscles and each plays a specific role in the movement of our eyes. CN IV controls our superior oblique muscles, which control intorsion, depression and abduction. 2,3 Loss of this muscle's function causes an upward deviation of the affected eye with a cyclotorsion that causes the patient to tilt their head away from the lesion. 2. Ptosis (pronounced toe-sis) is actually an abbreviation for the medical term blepharoptosis. Ptosis is a condition where one or both of the eyelids droop and obstruct vision. The cause may be anatomical, such as excessive skin (dermatochalasis) over the eyelids, traumatic, or neurological (damage to the third cranial nerve, myasthenia gravis, or Horner's syndrome for example) Neuropathy is a common complication of diabetes, in which cranial nerve palsies are rare and associated with long-standing poorly controlled type 2 diabetes. We report a case of a young patient with oculomotor nerve palsy as the presenting symptom of type 1 diabetes. A 36-year-old previously healthy Sudanese man was referred to our emergency department because of progressive diplopia of his.
Isolated third cranial nerve palsies in head trauma patients can be the result of direct or indirect damage to the oculomotor nerve. They are usually associated with severe head trauma. We reported a case of isolated oculomotor nerve palsy associated with minor head injury. No initial loss of consciousness was recalled. Computed tomography (CT), magnetic resonance imaging (MRI), and magnetic. Inspect the eyelids for evidence of ptosis which can be associated with: Oculomotor nerve pathology; Horner's syndrome; Neuromuscular pathology (e.g. myasthenia gravis) Eye movements. Briefly assess for abnormalities of eye movements which may be caused by underlying cranial nerve palsy (e.g. oculomotor, trochlear, abducens, vestibular nerve. Mononeuropathy means that only one nerve is damaged.This disorder affects the third cranial nerve in the skull. This is one of the cranial nerves that control eye movement. This type of damage may occur along with diabetic peripheral neuropathy.Cranial mononeuropathy III is the most common cranial nerve disorder in people with diabetes In addition to diplopia as a result of cranial nerve palsies, patients may present with abrupt onset of periorbital edema, headache, chemosis, ptosis and proptosis. CT scan can help reveal. -taste and sensation on posterior 1/3 of tongue, pharynx, & tonsils-carotid baroreceptor: blood pressure (carotid sinus)-carotid chemoreceptor: O2 & CO2 (carotid body) 3. Located on very top part of medulla and is right below the facial and vestibulocochlear branche
Sympathetic root. The sympathetic root of ciliary ganglion is one of three roots of the ciliary ganglion. It contains postganglionic sympathetic fibers whose cell bodies are located in the superior cervical ganglion.Their axons ascend with the internal carotid artery as a plexus of nerves, the internal carotid plexus.Sympathetic fibers supplying the eye separate from the carotid plexus within. Third cranial nerve palsy is a neurological condition that causes ptosis, double vision, eye turn (down and out), and in some cases, a dilated pupil. Congenital third nerve palsies may be related to birth trauma, infection, or a developmental abnormality Cranial Nerve III Palsy: Damage or disruption of this cranial nerve causes eyelid drooping because it innervates the levator. Typically, the ptosis is unilateral and the affected eye will present in a down-and-out position with a blown pupil 1. The concerning etiology in these cases is an aneurysm or tumor compressing the nerve, and should be. Third Cranial (Oculomotor) Nerve Disorders. Third cranial nerve disorders can impair ocular motility, pupillary function, or both. Symptoms and signs include diplopia, ptosis, and paresis of eye adduction and of upward and downward gaze. If the pupil is affected, it is dilated, and light reflexes are impaired Cranial nerve III has somatic and autonomic functions. Somatic nerves are homologous with ventral roots of spinal nerves. They originate from the basal plate and innervates the superior rectus, inferior rectus, medial rectus, and inferior oblique muscles. Additional symptoms include unilateral ptosis and decreased visual acuity..
This ptosis may relieve the unpleasant diplopia caused by the deviation of the affected eye. Lets look at the pathway of the oculomotor nerve. If we look at the brainstem from the side, through the temporal lobe, the oculomotor nerve nucleus lies at the level of the superior colliculus in the midbrain Ptosis is when the upper eyelid droops down over the eye. If the ptosis is severe, it may block vision. There is one main muscle that opens the eyelid (levator palebrae superioris). Another muscle helps the eye open even more (superior tarsal muscle). If there is a problem with either of these muscles or their nerves, ptosis can occur With unilateral third cranial nerve palsy (ie, oculomotor nerve palsy), the involved eye usually is deviated down and out (ie, infraducted and abducted), and there may be partial or complete ptosis Description and Pathology: The third cranial nerve controls most of the eyes movement including constriction of the pupil and maintenance of an open eyelid. The oculomotor nerve controls the eyes movement in conjunction with cranial nerves IV and V. As a result, when a pathology occurs in eye movement, all three cranial nerves III, IV
The oculomotor nerve is the third of the cranial nerves and arises from the midbrain.It is responsible for the movements of four of the six extraocular muscles, the other two being innervated by the trochlear and abducens nerves.. Gross anatomy Nuclei. There are two cranial nerve nuclei whose neurons contribute axons to the oculomotor nerve:. The oculomotor nucleus lies in the midbrain. Depression, abduction, and intortion, along with mydriasis and ptosis indicate palsy of which cranial nerve. Cranial Nerve III. Aneurysm of which cerebral artery often impinges on CN III. Aneurysm of Posterior Cerebral Artery. What is the Pupillary Light Reflex. the reflex that pupil has to light
The third cranial nerve, or oculomotor nerve, supplies several extraocular muscles as well as the ciliary muscle, sphincter pupillae, and levator palpebrae superioris .While there are several causes of third cranial nerve palsy, the most common have been identified as hypertension and diabetes mellitus .Coronavirus disease 2019 (COVID-19) is known to be primarily a viral infection affecting. Cranial nerve palsies often resolve themselves over a few months. If they do not, our focus is to treat the symptoms as well as the underlying cause. At Children's National, our treatment options include: Glasses to improve vision and eliminate double vision. Surgery on the eye muscles to realign the eyes and eliminate double vision and ptosis Cranial Nerve Treatment and Surgery Options. The first treatment approach for cranial nerve disorders is medication. Unfortunately, medicine does not always help treat these disorders. Many people experience breakthrough pain or suffer undesirable side effects. In such cases, UPMC surgeons usually recommend microvascular decompression surgery Overview. Differential diagnosis of cranial nerve lesions includes central and peripheral causes. Causes vary according to which cranial nerve is affected, and whether multiple cranial nerves are involved. See also Bulbar and pseudobulbar palsy
Sudden eyelid drooping, or ptosis, can have a variety of causes. Peripheral nerves that travel through the neck can be disrupted, causing eyelid drooping along with a constricted pupil and lack of sweating (triad of Horner's syndrome). The eyelid drooping could also be caused by an abnormality in the brain such as bleeding or a stroke 1. The orbicularis oculi muscle surrounds the circumference of the eye and serves to close both the upper and lower eyelid. It is innervated by the seventh cranial nerve (facial). 2. The levator palpebrae superiors muscle helps to raise the upper eyelid. It is innervated by the third cranial nerve (oculomotor). 3 Congenital third cranial nerve palsy. Congenital oculomotor palsy may be partial or complete and is thought be caused by either birth trauma or occur as a developmental abnormality. The presenting feature is ptosis, with varying degrees of ophthalmoplegia, consisting of an inability to fully depress, adduct and elevate This cranial nerve is difficult to evaluate and is only rarely evaluated in a clinical setting. miosis (pupillary constriction), enophthalmos (retraction of the globe), ptosis (narrowing of the palpebral fissure) and protrusion of the third eyelid. The most important diseases of the sympathetic and parasympathetic nervous system include.
Cause #3: Nerve Disorders. Cranial nerve and brain injuries due to any reason can cause ptosis as it affects the nerve supply to the muscles of the eyes and eyelids. Brain tumor, stroke, aneurysm and long-term diabetes may also cause this to happen Ptosis. drooping of the upper eyelid. Diplopia. an optical condition which an object is seen as being two. cranial nerve impairments ipsilateral to the site of the lesion motor control loss on side opposite of the lesion. Signs of cerebellar lesions. ipsilateral effects ataxia, asynergia, dysdiadokinesia, dysarthria, dysmetria, intonation. Key points • Blepharoptosis, or ptosis, refers to the abnormally low position of the upper eyelid in primary gaze. • Ptosis of the eyelid is usually secondary to involutional changes, or is congenital in nature, although a broader classification scheme has been developed ( Box 3.1 ). Box 3.1 Classification of ptosis by etiology Congenita Ptosis is a condition characterized by the drooping of the upper eyelid. While the symptoms of ptosis may seem pretty straightforward — it is in the title, after all — symptoms experienced in addition to a droopy eyelid could help indicate the source of the issue.. Ptosis symptoms. Symptoms of ptosis can be very mild and unnoticeable, or severe An expanding mass in the cavernous sinus can compress cranial nerves III, IV, V, and VI, thereby producing various degrees of cranial nerve palsy as the signs and symptoms 10). However, isolated third cranial nerve palsy with ptosis as the presenting sign of pituitary apoplexy is very rare
The eyelids protect the eye and help maintain the corneal tear film. Ptosis (drooping), retraction (abnormal elevation), facial weakness (causing insufficient eyelid closure), abnormal blinking (absent or excessive), and other abnormal eyelid and facial movements are the most important eyelid and facial nerve disorders in neuro-ophthalmology 3 Children with IIH may present with diplopia due to an associated nerve palsy, which usually resolves promptly with acetazolamide treatment, as seen in the presented case. 4 However, the involvement of four cranial nerves (II, III, VI and VII) in the setting of IIH presented here is an exception. Surgical treatment options for IIH include. Lab 10 - Cranial Nerve Nuclei and Brain Stem Circulation Cranial Nerve III-Oculomotor Nerve. Components of the Oculomotor Nerve include The somatic motor component of the oculomotor nerve innervates the levator palpebrae, the superior, medial and inferior recti, and the inferior oblique muscle.; The parasympathetic component of the oculomotor nerve consists of preganglionic parasympathetic.
Cranial Nerve III, IV, and VI - Oculomotor, Trochlear, Abducens. Cranial nerve III, IV, and VI (oculomotor, trochlear, abducens nerves) are tested together. Test eye movement by using a penlight. Stand 1 foot in front of the patient and ask them to follow the direction of the penlight with only their eyes What Are the Causes of Ptosis? Typically, Ptosis results from a weakening in the muscle responsible for raising the eyelid or from damage to the nerves that control the muscle. In some cases, Ptosis may simply be caused by loose skin on the upper eyelid. Sometimes, however, the reason for Ptosis is more troubling Definition (MSH) The 3d cranial nerve. The oculomotor nerve sends motor fibers to the levator muscles of the eyelid and to the superior rectus, inferior rectus, and inferior oblique muscles of the eye. It also sends parasympathetic efferents (via the ciliary ganglion) to the muscles controlling pupillary constriction and accommodation
Pathological MR were classified depending on MR aspect and location of the disease : Orbital lesion (15), cavernous sinus disease (9), lesion impairing the III rd cranial nerve (13), Horner's syndrome (16, with among them 13 internal carotid dissections ), others causes : 4 Correlation with clinical data were made Ptosis may be secondary to: a. paresis of a branch of cranial nerve III. b. hyperthyroidism. c. psoriasis. d. blepharitis. ANS: A Ptosis is caused by a congenital defect of the muscle around the eye controlled by cranial nerve III. Hyperthyroidism causes exophthalmos. Psoriasis is a skin condition. Blepharitis is inflammation of the eyelid Description. Twelve pairs of nerves (the cranial nerves) lead directly from the brain to various parts of the head, neck, and trunk. Some of the cranial nerves are involved in the special senses (such as seeing, hearing, and taste), and others control muscles in the face or regulate glands. The nerves are named and numbered (according to their. These are the structures responsible for elevating the lid and are most relevant to the development of ptosis. The levator muscle is a striated muscle innervated by cranial nerve III (oculomotor nerve) and extends from the lesser wing of the sphenoid to the level of Whitnall's ligament
Cranial mononeuropathy III is a nerve disorder. It affects the function of the third cranial nerve. As a result, the person may have double vision and eyelid drooping . Nearly half of patients with Guillain-Barré syndrome have cranial nerve involvement. However, isolated bilateral ptosis without ophthalmoplegia is a rare manifestation, and isolated unilateral ptosis without ophthalmoplegia in Guillain-Barré syndrome has not previously been reported in the literature Since cranial nerve III also controls the levator palpebrae superioris muscle, ask the patient to focus on a spot and observe the position of the eyelids. Note if ptosis, which is drooping of the upper eyelids, is present. Ptosis can be associated with lesions of the third nerve, Horner's syndrome, and neuromuscular diseases, such as myasthenia. Based on the clinical manifestations of ptosis, facial palsy, and trismus, we considered the possibility of primary cranial nerve palsy and Guillain-Barre syndrome as differential diagnosis in addition to tetanus. At her first presentation, primary cranial nerve palsy was strongly suspected because trismus was not observed
Painful cranial nerve III palsy. The cranial nerve III, known as the oculomotor nerve, is separated to two major components which are the outer parasympathetic fibres that innervate the ciliary muscles and the iris sphincter and the inner somatic fibres that supply the levator palpebrae superioris of the eyelid and four of the extraocular muscles (superior, middle, inferior recti and inferior. . The ptosis crutch is a nonsurgical option that involves adding an attachment to the frames of your glasses. This attachment, or crutch, prevents drooping by holding the eyelid in place Bilateral VII weakness. General. Definition: 2nd facial nerve paresis occuring within 30 days of 1st. Frequency: 0.3% to 2% of patients with facial paralysis. VII nerve lesions. Hereditary. Amyloidosis: Gelsolin. Melkersson syndrome. Möbius syndrome & Congenital facial paresis While these signs abated, intermittent fevers (up to 103.0 degrees Fahrenheit) were noted in the week prior to presentation. Physical examination revealed evidence of facial nerve (cranial nerve VII) deficits, with a right-sided ear droop, ptosis of the right eye, and deviation of the muzzle to the left (Image 2). Application of fluorescein.
The Cranial Nerves serve the sense organs, muscles and internal organs. The Cranial Nerves are represented with both Roman and Arabic numerals or a name, for example Cranial Nerve VI, CN VI, the 6th Cranial Nerve and Abducens Nerve all refer to the same nerve. Cranial nerves III through XII arise in the Brainstem Cephalic tetanus is defined as a combination of trismus and paralysis of one or more cranial nerves. Cranial nerves III, IV, VI, VII, and XII may be affected, but the facial nerve is most frequently implicated. A 64-year-old female visited hospital for left ptosis followed by facial palsy after a left forehead abrasion in a car accident Nerves. Congenital ptosis can also be the result of a neurologic dysfunction or a neuromuscular junction failure of the levator muscle. 33 The superior branch of the CN III innervates the levator palpebra superioris muscle and the superior rectus muscle. CN III is located in the midbrain and is composed of multiple subnuclei Oculomotor-abducens synkinesis (OCABSN) is an autosomal recessive disorder characterized by a specific anomaly of extraocular muscle movements involving the oculomotor nerve (cranial nerve III) and the abducens nerve (cranial nerve VI). The superior branch of CN3 innervates the levator palpebrae superioris muscle, which raises the eyelid, and. When the eyelids are abnormal, the examiner should look for the presence of an orbital syndrome, diplopia with abnormal extraocular movements, and pupillary abnormalities, and should determine whether the findings are unilateral or bilateral. 17.2 Ptosis. Ptosis can be either congenital or acquired. 17.2.1 Congenital Ptosis
On MRI of brain and orbit, the right third cranial nerve could not be visualised along with significant atrophy of extraocular muscles (EOMs) supplied by it . The patient was diagnosed to have congenital fibrosis of EOMs (CFEOM) in view of typical features of bilateral limitation of EOM movements with ptosis and severe atrophy of EOM supplied. Ptosis and cranial nerve IV palsy reveal juvenile myasthenia gravis. Optometry. 2009; 80(7):342-9 (ISSN: 1558-1527) Bodack MI. BACKGROUND: Ptosis and strabismus are 2 common presenting complaints of preschool-age patients. In both cases, these conditions can be benign and require no further workup -taste and sensation on posterior 1/3 of tongue, pharynx, & tonsils-carotid baroreceptor: blood pressure (carotid sinus)-carotid chemoreceptor: O2 & CO2 (carotid body) 3. Located on very top part of medulla and is right below the facial and vestibulocochlear branche Sympathetic root. The sympathetic root of ciliary ganglion is one of three roots of the ciliary ganglion. It contains postganglionic sympathetic fibers whose cell bodies are located in the superior cervical ganglion.Their axons ascend with the internal carotid artery as a plexus of nerves, the internal carotid plexus.Sympathetic fibers supplying the eye separate from the carotid plexus within.